Americans live shorter lives despite being much richer

Friday, August 28th, 2026

The reasons Americans live shorter lives despite being much richer than their peers are well-understood at this point, Cremieux explains:

The biggest contributor is that obesity is very bad for health, as countries get richer their citizens tend to get fatter, and America is the richest country.

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All Americans regardless of race are, in principle, exposed to a health care system of the same quality understood broadly, although there are gaps in access, but those access gaps don’t fit the order of the longevity gaps—as a close proxy, in 2024, the insurance rates go Asian > White > Black > Hispanic. These race results and the sex ones combine to produce a picture that cannot be explained by a simple health care-centric story. In fact, when it comes to things the health care system can plausibly intervene on, America is usually the highest-performing country—at least cross-sectionally—or it’s very nearly so. When it comes to lifespan and America’s lag despite its riches, look elsewhere than health care.

Comments

  1. Bob Sykes says:

    Although the obesity epidemic (at least two-thirds of everyone) explains much the the differences in death rates among Americans (and between Europeans and Americans), the basic cause is genetics. About 80 to 90 % of life expectancy is genetics, 5 to 10% diet, and 5 to 10% excercise and fitness.

  2. Isegoria says:

    Longevity definitely has a genetic component:

    Three decades ago a famous study of Danish twins found that our genes “only moderately” influence how long we’re likely to live. Longevity, the authors estimated, was about 25 percent heritable, meaning the remaining three quarters was determined by environmental factors and lifestyle choices, such as diet and exercise. Most subsequent studies found heritability to be somewhere in the 20 to 25 percent range, and 25 percent is now widely accepted. But a new study more than doubles it, suggesting lifespan may be more genetically fixed than we thought.

    The study, which was published today in Science, arrives at this dramatic increase by reframing how scientists think about longevity. Rather than lumping all deaths together, the researchers distinguish between two kinds: “intrinsic mortality” comes from built-in biological aging processes and genetic mutations, whereas “extrinsic mortality” comes from outside causes, such as accidents and infection. Early longevity studies analyzed groups of people who were born in a time of widespread extrinsic mortality. That skewed previous estimates of heritability, says Uri Alon, a systems biologist at the Weizmann Institute of Science in Rehovot, Israel, and senior author of the new paper.

    To sort out the effects of intrinsic versus extrinsic deaths on longevity heritability, he and his colleagues ran computer simulations of human mortality, calibrated using data from those previous twin studies. When they dialed extrinsic mortality down to zero, leaving only deaths caused by intrinsic aging processes, lifespan heritability roughly doubled.

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